Conclusion The mechanism of fluoride-resistant strain may not be the result of the variation of ATPase gene. 结论ATP酶基因改变不是变链菌株耐氟突变的机制。
Na+, K+- ATPase may also be responsible for pumping norepinephrine back into the sympathetic neurons to inactivate this neurotransmitter. 钠、、TP酶对去甲肾上腺素泵回交感神经元灭活这神经递质也有责任。
Abnormal Na transport across the cell wall due to a defect in or inhibition of the Na-K pump( Na+, K+- ATPase) or due to increased permeability to Na+ has been described in some cases of hypertension. 由于钠-钾泵(a+,K+-ATP酶)缺陷或抑制,或是由于对钠通透性的增高,导致跨细胞壁钠转运异常,这种情况已在一些高血压病例中加以阐述。